There are different causes of Osteoarthritis. Osteoarthritis usually happens gradually over time. Some risk factors that might lead to it include: Being overweight, Getting older, Joints injury, Joints that are not properly formed, a genetic defect in joint cartilage and Stresses on the joints from certain jobs and playing sports. It will pass from parent to child but the
Children that are born with FOP have missing joints or strange lumps near the joints. This usually occurs before ten years of age. This is a type of genetic disease where the bone growth progresses from the top downward, just as bones grow in fetuses. A child with FOP
It is caused by the absence of dystrophin, a protein involved in maintaining the integrity of muscle. Symptoms usually begin to appear between three and five years of age. The disorder progresses rapidly, starting with muscle weakness in the legs and pelvis, and eventually spreading to the arms, necks, and other areas. By the age of 12, most boys cannot walk and require the use of a wheelchair. They can also develop scoliosis and tightness in their joints.
It is the most common cause of hip pain in children pre-puberty, which causes arthralgia and arthritis secondary to a transient inflammation of the synovium of the hip. It affects children, ages 3-10, and males at double the rate of females (Whitelaw, 2013). Although transient synovitis is an idiopathic condition, it is most seen after a viral infection. It occasionally develops after getting a vaccine or from taking some medicines. The viral infection, vaccine, or medicine triggers a process that leads to an immune response that affects the joints.
Spinal Muscular Atrophy Spinal muscular atrophy or SMA for short is a neuromuscular disease. SMA represents a group of autosomal-recessive disorders, leading to muscle weakness and atrophy. This disorder is common and is passed on genetically to children by their parents. You cannot “catch” Spinal Muscular Atrophy by being around someone who has it. It is caused by progressive deterioration of the anterior horn cells of the spinal cord.
The diagnosis of progeria is based upon physical appearance (Gulli). Since affected babies may appear normal at birth a diagnosis can't be made until the age of two (Livneh). A sign at the age of two may be changes in skin or growth failure (Gulli). The skeleton of one with progeria is often hypoplastic or, underdeveloped (Gulli). Commonly progeria is easily diagnosed by changes to or altered bone structure, joints and cartilage.
The gene contains a member of the fibroblast-growth-factor receptor (FGFR3) family, which is shown in articular chonfrocytes. A missense mutation in the fibroblast growth factor receptor, on chromosome 4p, in humans, causes achondroplasia. Symptoms of achondroplasia are 1- Disproportionately short arms and legs, 2- Short fingers, 3- A large head with a prominent forehead, 4: A mid-face, 5- Short stature, and 6: Decreased muscle tone. Some children have delayed motor milestones, bowing of lower legs, and frequent ear infections. Intelligence and life span is not affected while having achondroplasia.
Recovery times are lengthy as mentioned and vary depending on the person, reason for the procedure, and what kind of care measures are taken after the procedure. In one statement it is “a general rule, children heal in half the time as it takes for adult patients. For example, when the desired goal is 1 and one-half inches of new bone growth, a child will wear the fixation device for three months; an adult will likely take 6 months to complete the process” (HSS, 2009). For both children and adult the obvious benefit is to alleviate problems associated with whatever condition called for the procedure in the first place be it physical relief or psychological. Interestingly, both children and adults can benefit from it in a social sense helping to alleviate depression-associated issues or feelings, having more confidence, and a general, improved well-being.
The disease is often detected in the first few weeks of life. Due to the buildup of lipids in the CNS, motor control, swallowing, and mental capabilities are impaired. In the very worst of cases most children do not live beyond two years, in mild cases some are known to live into their teen years. The trait is passed on when both parents carry a defective gene that moderates protein sphingomyelin. The likelihood of the child getting Farber’s is twenty five percent; there is a fifty percent chance that the child will carry the defective gene with no symptoms, a carrier.
There are many causes of pressure sores here are a few examples : * Being unable to move around easily due to old age or illness. * Weight loss - you may have less padding over bony areas. * Sliding down in a bed or chair - pressure on the skin cuts off blood supply because the skin is being pulled in different directions. * Friction or rubbing of the skin, for example against sheets. * Moist skin - for example, due to sweating or incontinence.