Multiple sclerosis Multiple sclerosis This booklet includes- The symptoms of Multiple Sclerosis The impact of Multiple Sclerosis The signs of Multiple Sclerosis What is multiple Sclerosis? Multiple Sclerosis is a disease that affects the brain and the spine, eventually it ends in not being able to control muscles, loss of vision, not being able to balance, and numbness. When you suffer from multiple sclerosis the brain and spine are damaged by the immune system, this is known as an autoimmune disease. The symptoms- The symptoms affect every person differently, they vary on how much damage has been done to the nerves. Some symptoms include: -visual Problems: the visual problems are one of the first to occur normally.
Depending on the complexity of the retinal detachment, various combinations of vitrectomy, buckle, laser and gas bubble may be used to repair the retina. Ataxia describes a lack of muscle control during voluntary movements, such as walking or picking up objects. A sign of an underlying condition, ataxia can affect movement, speech,
If left untreated, a MRSA skin infection can go progressively deeper into the body, infecting blood and organs. Read more: http://www.foxnews.com/health/2012/05/08/mrsa-antibiotic-resistant-bug-that-has-health-officials-worried/#ixzz2UnWmfNeQ When MRSA infects skin, a swollen, red area develops, and it is usually painful. MRSA skin infections may develop pus or weep other fluids. If left untreated, a MRSA skin infection can go progressively deeper into the body, infecting blood and
(Guest Josephinez, 2011) Multiple Sclerosis (MS) is an autoimmune disease that affects the central nervous system (brain and spinal cord). Most of the time, MS is diagnosed between the ages of 20 and 40, but it can be discovered at any age. The chance of getting MS, on average, is 1 in 750.While anyone can get Multiple Sclerosis, it is 2 to 3 times more common
The name is derived from the spongy holes that are only visible under a microscope that develop in the affected brain tissue. The risk of getting Creutzfeldt-Jakob disease is very low and usually affects people over the age of 60. It cannot be transmitted through coughing, sneezing, touching, or sexual contact. There are three ways that you can get Creutzfeldt-Jakob disease. You can get it spontaneously, and most
IVIG can potentially interfere with these autoantibodies as well as suppressing the response to proinflammatory cytokines, which are important in the mechanisms of CRPS pain and hyperalgia, in the peripheral and the central nervous systems. Pathophysiologic findings support a mechanism that may explain the results of this
In sickle cell anemia, the abnormal haemoglobin (Haemoglobin-S) sticks together when it gives up its oxygen to the tissues. These clumps cause red blood cells to become stiff and shaped like a sickle. It takes two copies of the sickle cell gene for the body to make the abnormal haemoglobin found in sickle cell anemia. Sickle-cell anemia is caused by a point mutation in the ß-globin chain of haemoglobin, replacing the amino acid glutamic
In the acute phase patients will experience psychotic episodes and have no perception of reality. In the stabilization phase a patient’s psychotic episodes are managed but they can relapse at any time. In the last and final stage, maintenance phase, the patient is stable but they have to take antipsychotic medicine in order to control their symptoms. The behavioral changes that the schizophrenic experiences can be categorized as positive or negative. Positive symptoms are hallucinations, delusions, hearing voices, thought insertion and thought removal, and somatic hallucinations.
Being female, women are about twice as likely as men are to develop MS. Having certain infections, a variety of viruses have been linked to MS, currently the greatest interest is in the association of MS with Epstein-Barr virus, the virus that causes infectious mononucleosis. Having a family history, if one of your parents or siblings has had MS. you have a 1 to 3 percent chance of developing the disease as compared with the risk in the general population, which is just a tenth of
The disease is thought to be caused by low levels of a chemical called dopamine, which activates the cells in our brain that let us move. While the cause of this neuronal death remains tentative, researchers have identified several cellular characteristics that are common in this disease and which appear to play a role in the neurons' degeneration. The principal among these is the presence of Lewy bodies in neurons of the substantia nigra, the brainstem, and other parts of the brain. It may obstruct with communication of nerve signals or other significant neuronal functions. Another cellular characteristic of Parkinson's is the presence of Lewy neurites — nerve fibers swollen with proteins.