To What Extent Is the Biological Approach an Adequate Explanation for the Initiation, Maintenance and Relapse of Smoking Behaviour?

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When the nicotine in a cigarette is inhaled this activates nicotinic receptors in the brain which leads to a release of dopamine in the NA. This creates a sense of pleasure however this sensation of the nicotine is rapidly broken done by enzymes in the liver and removed from the bloodstream so within hours this pleasure sensation is replaces by low moods and reduced concentration. According to the biological model people with certain genes may find it harder to stop behaviour once it has started. There is no single gene responsible for this, however a number of genes have been linked to playing a role. This has been shown in research done by Noble et Al in which a gene DRD2 responsible for the D2 dopamine receptor, to severe alcoholism. It was found that the A1 variant of this gene was present in more than two-thirds of deceased alcoholics. Although this research does not look at smoking behaviour, it suggests that this gene plays a role in the addictive behaviour and can also be generalised to smoking behaviour. There has also been additional evidence to support the biological model as an explanation for smoking addiction. Research has been done by Silverstein et al in which it was found that biological factors may influence a person’s first positive or negative experience with their first cigarette. For example whether they feel nausea, or choke. Although this does not explain why a person initially picks up a cigarette it does give us an explanation for why some people continue the initiation of smoking. Those with worse experiences due to the biological factors may be less likely to continue to start smoking. Hall et al also suggests that nicotine addiction is likely to involve multiple forms of genes that each have a small effect that interacts with each other and environmental factors. As there is this evidence to support the biological model this makes it

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