Ptsd Etiology Essay

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Posttraumatic Stress Disorder Etiology In the model presented in the article, A Diathesis-Stress Model of Posttraumatic Stress Disorder: Ecological, Biological, and Residual Stress Pathways, by McKeever and Huff (2002), shows many the many factors that contribute to posttraumatic stress disorder (PTSD). PTSD has been around for over a hundred years, and went by many names. Those diagnosed with it would often be considered inherently weaker and therefore be predisposed to pathological responses. DSM-III in 1980 was the first time the principal etiological factor of PTSD was the traumatic experience. Even though it was acknowledged that how severe the trauma was an influential factor, the question of why people developed PTSD was not heavily researched. The diathesis-stress model presented by McKeever and Huff attempts to answer that question. The proposed diathesis-stress model for PTSD takes into account the traumatic experience itself, the residual stress, as well as ecological and biological diatheses. The more ecological and biological stressors there are, the less severe the trauma would need to be to break the threshold and activate the diathesis. The residual stress acts as the catalyst to PTSD and it cannot happen without it. The more severe a person’s residual stress, the high risk they possess of breaking that threshold and developing PTSD. Ecological factors can be anything from amount the amount of social support to previous traumas. These could all be part of the ecological diathesis, though not always. Because residual stress is reserved for the traumatic event that spurs PTSD, other events are then put in the ecological category. Duncan et al. found that women with histories of child abuse were nearly five times more likely to have a lifetime history of PTSD and ten times more likely to currently be experiencing PTSD than women without histories
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