Approximately one third of American adults were believed to be obese in 2013. Obesity has many different effects, some are short term and others are long term, but all are negative. Some short term effects are that risk factors for cardiovascular disease immediately increase, which is known as high blood pressure and high cholesterol. This can also be a long term effect because it affects young people when they get older. Obesity can also cause diabetes, heart disease, joint problems, respiratory problems, cancer, metabolic syndrome, and psychosocial effects.
The obesity in childhood is also related to poor eating habits, lack of exercise, family history of obesity, medical illness, and depression or other emotional problems. Obesity can also end in serious health issues that can be hard to treat. Obesity and cardiovascular disease is in a close relation. The excess fat that circulates the blood that turns into plaque. The plaque accumulates around an artery to block the flow of blood.
This is measured in terms of the impact of being absent (absenteeism) from work or being less productive at work that a healthy person (presenteeism), and loss of productivity due to premature mortality caused by obesity. The indirect costs are associated with productivity losses (both absenteeism and presenteeism) and premature mortality is estimated to be $6.4 billion in 2008/2009. In addition to the costs of obesity related to healthcare, productivity and premature death, the burden of disease costs associated with obesity were estimated to be approximately $30 billion in 2008/2009. Notably, this is significantly larger than the other costs presented above and are important to consider, as this estimate captures not only the financial but also the social costs of obesity. Absenteeism was found to be 14 percent higher is obese employees compared with normal-weight employees in the working population.
Obesity, having excessive amounts of body fat, impairs health and has serious physical and mental consequences. (http://www.bmicharts.org, 2011) Causes of childhood obesity There is a variety of factors associated with causing childhood obesity. Although genetics can affect weight, evidence shows that lifestyle choices and environmental factors have the largest impact. Little physical movement and/or consuming too many calories can result in considerable weight gain. These sedentary lifestyles contribute greatly to childhood obesity.
This makes them directly correlated to the current trends in the prevalence of obesity. According to Friel (2009) inequity which results to social profiles has resulted to increased prevalence in obesity. The affluent, with good income spend much eating junk foods and doing less exercise. The underprivileged on the other hand are bound by illiteracy, poor social amenities, retrogressive cultures and unemployment. This makes both groups vulnerable to obesity as they indulge in unplanned and bad eating habits.
6 Issue Issue 7, p3-7. 5p. Article This article discusses the impacts of medical costs due to the obese and overweight population. It also discusses the health issues caused by obesity. The obesity epidemic is affecting adults as well as children.
Our genetic makeup affects an extensive range of chemical functions in our body and may affect your weight in various ways including your metabolism, hormones and body fat disposition. While there is widespread acceptance that hereditary factors might predispose to human obesity, it is frequently assumed that such factors would influence metabolic rate or the selective partitioning of excess calories into fat. However, it is notable that, thus far, all monogenic defects causing human obesity actually disrupt hypothalamic pathways and have a profound effect on satiety and food intake. To conclude, the evidence we have to date suggests that the major impact of genes on human obesity is just as likely (or perhaps more likely) to directly impact on hunger, satiety and food intake rather than metabolic rate or nutrient partitioning. At the risk of oversimplification, it seems that from an aetiological/genetic standpoint, human obesity appears less a metabolic than a neuro-behavioural disease.
This medical term is called hyperglycemia. Family history and genes also play an important role in this type of diabetes. Low activity level, an excess of body weight around the waist, and a poor diet, increases the risk of acquiring this devastating disease. Your health care provider may suspect you have diabetes if your blood glucose levels are higher than 200 mg/dL. Symptoms of this type of diabetes are: fatigue, hunger, increased thirst, and increased in urination.
And it is not surprising that childhood obesity is a good indicator for the likelihood of obesity into adulthood. What is surprising, however, as Galvez (2003) points out, is that "studies on obesity consistently report a higher prevalence of obesity in African Americans and Mexican Americans compared with the white, non-Hispanic population" (p. A684). The etiology of childhood overweight is multifaceted, but is seemingly always tied to socioeconomic factors. Poor nutritional choices and a sedentary lifestyle cause obesity, but it is societal stressors that lead to these inadequate choices. Such stressors include increased exposure to television advertisements for unhealthy foods, unsafe neighborhoods which inhibit outdoor play and exercise, and limited availability of healthy, fresh foods in low socioeconomic residential areas (Galvez 2003).
This means that they have little time to work out to burn off the fatty foods that they consume on a daily basis. Now there has been a confusing with the term obesity and overweight. Overweight refers to excess body and this includes all tissues. With obesity on the increase so much lately it has appear to show that it could be genetic. Most American adults grown more than an inch over and gain twenty five more pounds than normal since the early sixties, according to the National Center for Health Statistics.