Diathesis-Stress and Ptsd Essay

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The diathesis–stress model will be utilized to explain why some individuals are more at risk (or predisposed) for developing Posttraumatic Stress Disorder (PTSD) than others via an analysis of current research. This connection can lead to a greater understanding about the etiology of PTSD and accordingly direct therapeutic options. As PTSD is an over-riding issue in therapy, theorized as having connections to many psychiatric diagnosis ranging from Borderline-personality Disorder (Ball and Links, 2009) to psychosis (Larkin and Read, 2008), this analysis can be invaluable clinically, and possibly save financial and resource costs by looking at the roots of untreated trauma which may manifest itself in manners in addition to PTSD. The diathesis-stress model can be conceptualized as a multi-causal developmental model, proposing that during maturity varying risk factors interrelate with stressors (and protective factors), accordingly contributing to development and sometimes psychopathology (Masten, 2001). Post-traumatic stress disorder (or PTSD) is classified as an anxiety disorder, typified by anxiety-related encounters and physiological responses that develop after exposure to a psychologically traumatic incident. Categorically, features persist for longer than 30 days and are often disruptive to multiple aspects of life (Rothschild, 2000). The diathesis-stress model of PTSD demonstrates that a traumatic incident can serve as the primary stressor and thus, the activator of PTSD symptomology. LITERATURE REVIEW: Developmental Models Using a Diathesis-stress model, some researchers feel that even pre-birth traumatic experience in-utero can create vulnerabilities and have predictive effects on later mental health functioning, as Froukje and Broekman (2011) have recently postulated. However, Froukje and Broekman caution making direct claims of causality from

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