How would the resistance of Cari’s airways be affected by excess mucus and fluid in her lung? The excess mucus and fluid in the lungs would increase the resistance of Cari’s airways because of the small diameter the buildup would cause and the fluid would have some alveoli underwater where they can’t function properly causing more friction from the buildup. 5. How would Cari’s lung compliance be altered as her alveoli fill with fluid due to pneumonia? Cari’s lung compliance would increase from trying to force gases in and out of the alveoli that are filled with fluid.
The extra mucus and fluids put extra pressure on the lungs. E) How would Cari’s lung compliance (the effort required to expand the lungs) be altered as her alveoli fill with fluid due to pneumonia? Cari’s lung compliance would increase from trying to force gases into and out of the alveoli, which are
Cholesterol is essential for healthy cells, but if there is too much in the blood it can lead to CAD. Cholesterol is carried in the blood stream by molecules called lipoproteins. Cholesterol is made from eating fatty foods, excess alcohol and caffeine etc. Which means that the more of these you eat the more likely you are to suffer from CAD 3) Carbon monoxide, nicotine, and other substances in tobacco smoke can promote atherosclerosis and trigger symptoms of coronary artery disease. Smoking: * Causes the platelets in your blood to clump together easily by making your blood cells more "sticky" and more likely to form clots.
How was the diagnosis of ARDS made? How important is the medical history? Once again in order to understand why a diagnosis of ARDS was made we must first understand what ARDS is. ARDS is acute respiratory distress syndrome and it come about when fluid builds up in the tiny, elastic air sacs called alveoli in your lungs. More fluid in your lungs means less oxygen can reach your bloodstream.
The invading organism causes symptoms, in part, by provoking a large immune response in the lungs. The capillaries become leaky, and protein-rich fluid seeps into the alveoli. This results in a less functional area for oxygen-carbon dioxide exchange. The patient becomes relatively oxygen deprived, while at the same time retaining large amounts of carbon dioxide. During this process, mucus production is increased.
INTRODUCTION Respiratory failure is a sudden and life-threatening deterioration of the gas exchange function of the lung. It exists when the exchange of oxygen for carbon dioxide in the lungs cannot keep up with the rate of oxygen consumption and carbon dioxide production by the cells of the body. DEFINITION Acute respiratory failure (ARF) is defined as a fall in arterial oxygen tension (PaO2) to less than 50 mm Hg (hypoxemia) and a rise in arterial carbon dioxide tension (PaCO2) to greater than 50 mm Hg (hypercapnia), with an arterial pH of less than 7.35. Respiratory failure is inadequate gas exchange by the respiratory system, with the result that levels of arterial oxygen, carbon dioxide or both cannot be maintained within their normal ranges. The normal reference values are: oxygen PaO2 greater than 80 mmHg (11 kPa), and carbon dioxide PaCO2 less than 45 mmHg (6.0 kPa).
Another complication that occurs is aspiration of gastric contents, especially if emesis occurs during noninvasive ventilation. To avoid this complication noninvasive ventilation shouldn’t be administered in patients with ongoing emesis or hematemesis. Complications of both noninvasive and invasive ventilation are Barotrauma and Hypotension. In Barotrauma (there is significantly less risk with noninvasive ventilation), and there is Hypotension, related to positive intrathoracic pressure which can be supported with
Overproduction of CSF Intracranial pressure rises if production of CSF is more than absorption. One possible cause is that CSF may be overproduced. In this case resistance to CSF flow, or venous sinus pressure may be increased as a result. CSF production will decrease as ICP rises. The brain compensates by absorption of CSF across ventricles.
Some of the causes of lung injury include the following: •prematurity - the lungs, especially the air sacs, are not fully developed •low amounts of surfactant (a substance in the lungs that helps keep the tiny air sacs open) •oxygen use (high concentrations of oxygen can damage the cells of the lungs) •mechanical ventilation - the pressure of air from breathing machines, suctioning of the airways, use of an endotracheal tube (ET tube - a tube placed in the trachea and connected to a breathing machine) Who is affected by chronic lung disease? Chronic lung disease can develop in premature babies who have had mechanical ventilation (breathing machine). Risk factors for developing CLD include: •birth at less than 30 weeks gestation •birth weight less than 1,000 (less than 2 pounds) to 1,500 grams (3 pounds 5 ounces) •hyaline membrane disease - lung disease of prematurity due to lack of surfactant that does not show the usual improvement by the third or fourth day. •pulmonary interstitial emphysema (PIE) - a problem in which air leaks out of the airways into the spaces between the small air sacs of the lungs. •patent ductus arteriosus (PDA) - a connection between the blood vessels of the heart and lungs that does not close as it should after birth.
These should only be prescribed by doctors. I do not wish to explain too much for fear that many may try to purchase these products over the counter. These medications are meant to be used for short periods – and drinkers must not consume alcohol while taking these medications. A common example is chlordiazepoxide, which has a calming effect to reduce the irritability, shakiness, and mood swings associated with quitting drinking. Another example is disulfiram (Antabuse).