D. Both Joseph’s instructions he needs to repair his body and his predisposition for vascular disease are both contain in Mitochondrial DNA. E. ATP was involved in the homeostatic imbalances of Joseph’s heart attack. The lack of ATP caused his cells to start dying. It also affected the plasma membrane pumps and the special calcium ATPases stopped moving the calcium from the cytosol to the endoplasmic reticulum. F. Reestablishing oxygen
B. Assuming Joseph’s heart has stopped, what cellular processes and membrane functions are going to be affected by the loss of oxygen, blood glucose, and waste removal? As time passed and Joseph lost oxygen, blood glucose and waste removal the cells continued to work which used up the ATP at a faster rate causing carbon dioxide levels to rise and pH to drop, which then caused the mitochondria to lack the oxygen and glucose that is required to make the ATP because of this the cells started dying. Plasma membrane was shut down by the active transport pump. Sodium began leaked into the cells and potassium exited.
C. Which intercellular organelles have membranes as part of their structure? 1. Mitochondrion: consists of an outer and inner membrane 2. Golgi complex: consists of 3-20 membranous sacs called cisternae 3. Endoplasmic reticulum: membranous networks of flattened sacs of tubules How would the breakdown of the membranes of these structures affect the function of Joseph’s heart cells?
Cellular Metabolism Case Study Part 1: 1. Given the autopsy report the massive cell death in the kidneys, lungs, thymus, and heart indicate that the damage occurred at the cellular level, the parts of cellular respiration that occur in the mitochondrion. More specifically, the poison directly affected the mitochondrion and in doing so it suggests that cellular respiration was disrupted. 2. The cellular process that would be impaired by arsenate would affect the cellular process of glycolysis which occurs in the cytosol of a cell.
Two ATP molecules are gained in this stage. The transition stage takes place in the mitochondria. The pyruvate is combined with NAD+ to form NADH and acetyl coenzyme A molecules. In citric acid cycle stage the hydrogen atoms are removed from the acetyl coenzyme A molecules to use the electrons to create ATP. Eventually, all that is left of the acetyl coenzyme A molecules is carbon, which combines with the oxygen to from carbon dioxide that is emitted as a waste product.
A human body consists of two functioning CFTR gene, and when neither gene functions efficiently, cystic fibrosis is developed, and hence has autosomal recessive inheritance. Inevitably, cystic fibrosis affects various cellular processes in a human body such as RNA/DNA replication, protein synthesis, and respiration.
Effect of Venturicidin on Oxygen Evolution Venturicidin is a powerful inhibitor of the chloroplast ATP synthase, interacting with the CFo part of the enzyme and blocking proton passage through the CFoCF1 complex. How would venturicidin affect oxygen evolution in a suspension of well-illuminated chloroplasts? Would your answer change if the experiment were done in the presence of an uncoupling reagent such as 2,4-dinitrophenol (DNP)? Explain. Answer Oxygen evolution requires continuing passage of electrons through PSII.
It works by affecting the electron transport chain of cells. There are several ways that one can succumb to death by this poison, ingestion, inhalation, and even absorption through the skin. Firstly, in order to understand how Zyklon B causes death in cells, one must understand how the cells work normally when they are healthy. Cells need energy to grow and maintain their functions. In cells, a majority of energy is carried in the form of a transport molecule called Adenosine
Think about it. The red blood cell's job is to carry oxygen. Mitochondria use oxygen to make ATP. If red blood cells had mitochondria, then some of the oxygen would be constantly used up. So -- no mitochondria either.
The destruction of beta cells cause Type I diabetes. The cause of the beta cells' destruction has yet to be determined, but scientists believe that it is caused by something that has gone wrong with the body's immune system. In Type II diabetes the problem is that the beta cells do not produce enough insulin to meet the needs of the body. Besides to this problem, the cells in the body does not react appropriately to the available insulin in the body because they cannot admit glucose through the cell membranes. Type II diabetes is caused by multiple factors.